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In: Principles of Diabetes Mellitus: Third Edition

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We also demonstrate intramolecular Friedlnder macrocyclization reactionssufficiently mild to be employed on unprotected and in vitro-translated peptidesthat embed a quinoline pharmacophore directly within the peptide backbone

In particular the stimulation of beta cell function and inhibition of glucagon secretion were important, since insufficient insulin secretion and hypersecretion of glucagon were at that time emerging as the key defects underlying type 2 diabetes (63, 64)

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